Diabetes and Candida
Candida is just one of many conditions that Diabetes can bring about, and diabetic women are particularly susceptible to any yeast infection. An elevated blood sugar level feeds the Candida yeast, allowing it grow quickly and dominate the other microorganisms in your gut. Here is some background and the story:
I just purchased and read a very intriguing text by Doug Kauffman called “Infectious Diabetes“. His premise in the book is that diabetes could probably be a fungal infection and given the right “terrain” manifest itself into diabetes. Kauffman was primarily a nutritionist and he found in his early work that prescribing patients with obvious fungi problems like toe nail and vaginal infections, a low carb diet with antifungal medication often proved very beneficial. He also noticed that many of these same patients were diabetic and, accordingly, their diabetes vastly improved at the same time. He found that the low carb diet and antifungal medication could often drop a patients blood sugar down to normal levels and keep them there, but as soon as they went back to their old diet and stopped antifungal medications, the old problems came back! He was excited that he may have found a unique break through for diabetics and told some MDs which he was working with. They immediately discouraged him from further study basing their opinion on the status quo–that diabetes was never treated by low carb diets (well in the 1970s) and antifungal medication (even now).
One of the main causes of diabetes as proposed by the American Diabetes Assoc is that people are over-weight and they eat too much. Kauffman asks, well if 20-30% of the American people are obese, why do not all of the obese people develop diabetes? We are missing 60-110 million diabetic cases in the rest of the obese population. He reasons that not all obese people get diabetes and 10% of type 2 diabetics are NOT over weight. He also finds the genetic angle to not hold water of which I will not go into in this post. His only logical conclusion is that diabetes is a fungi infection with the mycotoxins they produce.
Fungi is every where. We cannot avoid them. Often antibiotic use can open up our immune system to terrible fungi infections. There is also very real world problem of steroid induced diabetes, Streptozotocin (a fungi mycotoxin) induced diabetes, etc, etc. Might I add here that these and others can be linked to fungi infections with the main stream seemingly oblivious to this connection. Could not steroid usage stimulate fungi infections or statins which are derived from fungi mycotoxins also suggest that diabetes may be fungi based? Kauffman points out that there is evidence linking the drinking of cows milk to type 1 diabetes in children and he suggests it is the mycotoxin in the fat cells of milk that may be the link. The USDA does not screen for mycotoxins and they can be found any where in feed stuffs.
History of the Fungi/diabetic link:
1954………..uric acid can cause lab animals to develop diabetes
1963……….it is found that sacchromyces yeast (a fungus) produces uric acid.
1976……….Cryptococcus fungi is found in the islets of langerhans cells of two diabetic dying children. Later studies, Cryptococcus is injected into lab animals resulting in similar results. Cryptococcus also produces uric acid.
1990……….Type 1 diabetes was reportedly cured by using cyclosporin A an antifungal.
1973………it was shown that mutton is loaded with mycotoxins (a fungal toxin)
1981………a study of preg Icelandic women who ate cured mutton close to their pregnancy gave birth to babies with diabetes.
1980………alloxan (mycotoxin) damages insulin producing beta cells
1981……….Stremtozotocin, a mycotoxin (a fungal toxin) was shown to produce diabetes in lab animals.
1990………..lab rats on a 10% brewers yeast diet developed diabetes
1973……….common to see cancer patients treated with mycotoxins develop diabetes.
1990………lab animals were induced into diabetes with L-asparaginase
Kauffman goes on to point out that a corn mycotoxin that is commonly found on corn may be one reason why there seems to be a diabetic epidemic in Latin America. Asperigillus niger is another fungi that is found in many foodstuffs, particularly peanuts and corn. It generates oxalic acid in large amounts which can inhibit our cells that convert carbohydrates into energy leading to high blood sugar.
Sulfa drugs are commonly used to control blood sugar in diabetics by stimulating the pancreas. It is not normally recognized that sulfa drugs are also antifungals. The use of statin drugs have been shown to coincidentally lower the risks of producing diabetes. Antifungal means anticholesterol! The anti-cholesterol drugs were discovered accidentally because researchers were looking for antifungal medications! They found the statin group work against fungi because they inhibit an enzyme that our bodies need to process cholesterol. Fungi depend on cholesterol for survival Stopping cholesterol production in the fungi is an effective antifungal mechanism. Statin drugs are fungally derived and are mycotoxins. Fungi can produce cholesterol as well, so the ability of statin drugs to inhibit the enzyme and kill fungi at the same time may be the complex mechanism for statins,,,,,,,not simply a lowering of cholesterol. In short, Kauffman suggests that high cholesterol in the human may also be fungi related.
That’s enough of the Kauffman theories for now. I just want to suggest that Turmeric does have antifungal properties and its diabetic prevention properties may work on this mechanism! I also seemed to have stopped diabetes from manifesting in my dog last summer by the use of a anti-microbial. Food for thought!
Removing the fungus itself is a good first step. Then work to deal with why it is being produced in the first place. Lufenuron is the first step toward a healthier life.